Fig. 30. — Anthracosis — coal-miner's lung — showing excessive accumulation of carbon pigment in the lymphatic spaces around the vessels of the lung. (X 50 diam.)

Fig. 36. — Polylobular cirrhosis, or " Gin-drinker's Liver, " showing well-formed fibrous overgrowth which has divided up the liver tissue into irregular masses and caused atrophic and degenerative changes in the liver cells. (X 24 diam.)

Fig. 32.-Fibroblastsin young temporary granulation tissue. These are spindle shaped and have long processes. It is from these cells the permanent fibrous tissue is formed. (X 400 diam.)

Fig. 3 1 .—Cells from inflammatory exudate showing active phagocytosis. The muno-nuclcar cells are ingesting and digesting many of the polymorpho-nuclear leukocytes. Note that those phagocytic cells are pushing out protoplasmic processes (pseudopodia) by which j, they grasp their victims. (X 1000 diam.)

Fig. 33. — Healing abscess showing a wall of young cellular and vascular granulation tissue, which separates the pus area (top of Fig.) from the muscle fibres seen at lower part of Fig. (X 60 diam.)

Fig. 37.— Chronic interstitial myocarditis, showing the muscle fibres in the heart wall being separated and becoming atrophied by a slow fibrous overgrowth of the connective tissue. (X 300 diam.)

Fig. 35. — Scar tissue in a healed wound. Note the disappearance of blood-essels and that the cellular character has diminished — the fibroblasts having now developed into well formed fibrous tissue. (X 200 diam.)

Fig. 34. — Granulation tissue showing the character and relation of the cellular elements to the new blood-vessels in the young temporary tissue. (X

200 diam.)